Pediatric Neuro-Oncology Tumor Board Quarterly Series_NTRK_Recording_Edited.mp4
Epigenetic loss of BAI1 expression in cerebellar granule neuron precursors inactivates the p53 tumor suppressor and facilitates medulloblastoma formation in the cerebellum, Erwin Van Meir
Targeting H3.3G34R/V re-wiring of the epigenome in pediatric glioblastoma of children and young adults, Lynn Bjerke
Phase 1/2 study of DSP-7888 in pediatric patients with malignant glioma, Hiroyuki Fujisaki
Elucidating molecular pathogenic mechanisms of the histone H3.3 G34R mutation in pediatric high-grade gliomas, Santiago Haase
ATRX loss confers enhanced sensitivity to combined PARP inhibition and radiotherapy in pediatric glioblastoma models, Janat Fazal-Salom
State-of-the-art Classification and Molecular Diagnostics of Childhood Brain Tumors
The ABCs of Chromatin Biology and Biochemical Insights Into the Mechanism of Histone Mutations
Pediatric Neuro-Oncology Tumor Board Quarterly Series_NTRK_Recording_Edited.mp4
Peds Tumor Board_ETMR_Recording_gallery_2844x1600.mp4
Pediatric Neuro-Oncology Tumor Board Quarterly Series_NTRK_Recording_Edited.mp4
Epigenetic loss of BAI1 expression in cerebellar granule neuron precursors inactivates the p53 tumor suppressor and facilitates medulloblastoma formation in the cerebellum, Erwin Van Meir
Epigenetic loss of BAI1 expression in cerebellar granule neuron precursors inactivates the p53 tumor suppressor and facilitates medulloblastoma formation in the cerebellum, Erwin Van Meir